(Q47071944)

5 January 2018

title

Drosophila fragile X-related gene regulates the MAP1B homolog Futsch to control synaptic structure and function (English)

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5 January 2018

0 references

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GOA release 2020-03-11

Futsch Dmel_CG34387

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GOA release 2020-03-11

author

Gerald M. Rubin

7

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5 January 2018

Robert Renden

4

object named as

Renden RB

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23 October 2018

Zhang YQ

1

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5 January 2018

Bailey AM

2

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5 January 2018

Matthies HJ

3

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5 January 2018

Smith MA

5

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5 January 2018

Speese SD

6

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5 January 2018

Broadie K

8

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5 January 2018

language of work or name

English

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publication date

1 November 2001

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5 January 2018

published in

Cell

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5 January 2018

volume

107

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5 January 2018

issue

5

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5 January 2018

page(s)

591-603

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5 January 2018

FMR1 Protein: Conserved RNP Family Domains and Selective RNA Binding

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1 reference

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Human and murine FMR-1: alternative splicing and translational initiation downstream of the CGG-repeat

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Purified recombinant Fmrp exhibits selective RNA binding as an intrinsic property of the fragile X mental retardation protein

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Abnormal dendritic spines in fragile X knockout mice: maturation and pruning deficits

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The FMR-1 protein is cytoplasmic, most abundant in neurons and appears normal in carriers of a fragile X premutation

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The fragile X mental retardation protein is a ribonucleoprotein containing both nuclear localization and nuclear export signals

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FMRP associates with polyribosomes as an mRNP, and the I304N mutation of severe fragile X syndrome abolishes this association

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Fragile X mental retardation protein: nucleocytoplasmic shuttling and association with somatodendritic ribosomes

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Analysis of neocortex in three males with the fragile X syndrome

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Drosophila Futsch/22C10 is a MAP1B-like protein required for dendritic and axonal development

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The central complex of Drosophila melanogaster is involved in flight control: studies on mutants and mosaics of the gene ellipsoid body open

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Abnormal dendritic spine characteristics in the temporal and visual cortices of patients with fragile-X syndrome: a quantitative examination.

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Understanding the molecular basis of fragile X syndrome

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The fragile X mental retardation protein is associated with ribosomes

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Evidence that fragile X mental retardation protein is a negative regulator of translation

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The fragile X mental retardation protein inhibits translation via interacting with mRNA

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Abnormal development of dendritic spines in FMR1 knock-out mice

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(Over)correction of FMR1 deficiency with YAC transgenics: behavioral and physical features

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Genetic studies in Drosophila: vesicle pools and cytoskeleton-based regulation of synaptic transmission

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Integrin-mediated regulation of synaptic morphology, transmission, and plasticity.

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7 January 2021

Drosophila Futsch regulates synaptic microtubule organization and is necessary for synaptic growth

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7 January 2021

The fragile X mental retardation protein binds specifically to its mRNA via a purine quartet motif

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7 January 2021

The protein product of the fragile X gene, FMR1, has characteristics of an RNA-binding protein

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7 January 2021

FMRP is associated to the ribosomes via RNA.

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7 January 2021

Differential expression of FMR1, FXR1 and FXR2 proteins in human brain and testis

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7 January 2021

Characterization and localization of the FMR-1 gene product associated with fragile X syndrome

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7 January 2021

Identification of a gene (FMR-1) containing a CGG repeat coincident with a breakpoint cluster region exhibiting length variation in fragile X syndrome

1 reference

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7 January 2021

Characterization of dFMR1, a Drosophila melanogaster homolog of the fragile X mental retardation protein

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7 January 2021

Fragile X mental retardation protein is translated near synapses in response to neurotransmitter activation

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7 January 2021

10.1016/S0092-8674(01)00589-X

Identifiers

DOI

1 reference

5 January 2018

1 reference

5 January 2018