(Q44952104)

7 December 2017

title

Glycine protects cardiomyocytes against lethal reoxygenation injury by inhibiting mitochondrial permeability transition (English)

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Antonio Rodríguez-Sinovas

7 December 2017

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4

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7 December 2017

Marisol Ruiz-Meana

1

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7 December 2017

Pilar Pina

2

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7 December 2017

David Garcia-Dorado

3

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7 December 2017

Ignasi Barba

5

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7 December 2017

Elisabet Miró-Casas

6

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7 December 2017

Maribel Mirabet

7

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7 December 2017

Jordi Soler-Soler

8

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7 December 2017

language of work or name

English

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publication date

24 June 2004

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7 December 2017

published in

Journal of Physiology

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7 December 2017

volume

558

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7 December 2017

issue

Pt 3

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7 December 2017

page(s)

873-882

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Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

7 December 2017

cites work

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Protection of ATP-depleted cells by impermeant strychnine derivatives: implications for glycine cytoprotection

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Contributions of mitochondria to animal physiology: from homeostatic sensor to calcium signalling and cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Protection by glycine against hypoxic injury of rat hepatocytes: inhibition of ion fluxes through nonspecific leaks

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

The mitochondrial KATP channel as a receptor for potassium channel openers.

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Calcium-dependent opening of a non-specific pore in the mitochondrial inner membrane is inhibited at pH values below 7. Implications for the protective effect of low pH against chemical and hypoxic cell damage

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Protection of hearts from reperfusion injury by propofol is associated with inhibition of the mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Mitochondrial permeability transition: a common pathway to necrosis and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

The mitochondrial permeability transition in cell death: a common mechanism in necrosis, apoptosis and autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Glycine blocks opening of a death channel in cultured hepatic sinusoidal endothelial cells during chemical hypoxia

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Pre-treatment with the Na+/H+ exchange inhibitor cariporide delays cell-to-cell electrical uncoupling during myocardial ischemia.

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Cariporide preserves mitochondrial proton gradient and delays ATP depletion in cardiomyocytes during ischemic conditions

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Amino acid release during volume regulation by cardiac cells: cellular mechanisms.

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

The mitochondrion in apoptosis: how Pandora's box opens

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

Glycine protection of PC-12 cells against injury by ATP-depletion

21 January 2018

1 reference

https://api.crossref.org/works/10.1113%2FJPHYSIOL.2004.068320

The permeability transition pore signals apoptosis by directing Bax translocation and multimerization.

21 January 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1665014

The mitochondrial permeability transition pore and its role in cell death

3 November 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1665014

Intravenous glycine improves survival in rat liver transplantation.

3 November 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1665014

Development of porous defects in plasma membranes of adenosine triphosphate-depleted Madin-Darby canine kidney cells and its inhibition by glycine.

3 November 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1665014

Glycine minimizes reperfusion injury in a low-flow, reflow liver perfusion model in the rat.

3 November 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1665014

Amino acid loss during volume regulatory decrease in cultured chick heart cells

3 November 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1665014

Mitochondrial dysfunction and cytoskeletal disruption during chemical hypoxia to cultured rat hepatic sinusoidal endothelial cells: the pH paradox and cytoprotection by glucose, acidotic pH, and glycine

3 November 2018

1 reference

12 December 2020

Mitochondrial ATP-sensitive K+ channels modulate cardiac mitochondrial function

1 reference

12 December 2020

Alterations of Na(+) homeostasis in hepatocyte reoxygenation injury

1 reference

12 December 2020

Haemodynamic effects of irrigating fluids studied by Doppler ultrasonography in volunteers

1 reference

12 December 2020

10.1113/JPHYSIOL.2004.068320

Identifiers

DOI

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7 December 2017

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