(Q43722709)

English

Repeated intermittent administration of psychomotor stimulant drugs alters the acquisition of Pavlovian approach behavior in rats: differential effects of cocaine, d-amphetamine and 3,4- methylenedioxymethamphetamine ("Ecstasy").

scientific article published in July 2001

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Europe PubMed Central

PubMed publication ID

26 November 2017

Repeated intermittent administration of psychomotor stimulant drugs alters the acquisition of Pavlovian approach behavior in rats: differential effects of cocaine, d-amphetamine and 3,4- methylenedioxymethamphetamine ("Ecstasy"). (English)

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

James David Jentsch

2

object named as

Jentsch JD

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Europe PubMed Central

PubMed publication ID

26 November 2017

author name string

Taylor JR

1

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Europe PubMed Central

PubMed publication ID

26 November 2017

language of work or name

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publication date

1 July 2001

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Europe PubMed Central

PubMed publication ID

26 November 2017

Biological Psychiatry

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Europe PubMed Central

PubMed publication ID

26 November 2017

50

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Europe PubMed Central

PubMed publication ID

26 November 2017

2

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Europe PubMed Central

PubMed publication ID

26 November 2017

137-143

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

The biological, social and clinical bases of drug addiction: commentary and debate.

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Differential effects of excitotoxic lesions of the basolateral amygdala, ventral subiculum and medial prefrontal cortex on responding with conditioned reinforcement and locomotor activity potentiated by intra-accumbens infusions of D-amphetamine.

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The tonic/phasic model of dopamine system regulation: its relevance for understanding how stimulant abuse can alter basal ganglia function

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Repeated d-amphetamine enhances stimulated mesoamygdaloid dopamine transmission

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Enhanced conditioned inhibition following repeated pretreatment with d-amphetamine

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Double dissociation of the behavioural effects of R(+) 7-OH-DPAT infusions in the central and basolateral amygdala nuclei upon Pavlovian and instrumental conditioned appetitive behaviours

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Enhanced acquisition of discriminative approach following intra-amygdala d-amphetamine

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Role for dopamine in the behavioral functions of the prefrontal corticostriatal system: implications for mental disorders and psychotropic drug action

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A role for sensitization in craving and relapse in cocaine addiction

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Reinstatement of drug-seeking behavior produced by heroin-predictive environmental stimuli

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Molecular and Cellular Basis of Addiction

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Conditioning factors in drug abuse: can they explain compulsion?

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Basal forebrain cholinergic lesions enhance conditioned approach responses to stimuli predictive of food.

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Disconnection of the anterior cingulate cortex and nucleus accumbens core impairs Pavlovian approach behavior: further evidence for limbic cortical-ventral striatopallidal systems

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Increasing effects of repetitive cocaine administration in the rat

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Relationship between reward-enhancing and stereotypical effects of psychomotor stimulant drugs

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Extinction and recovery of cocaine self-administration following 6-hydroxydopamine lesions of the nucleus accumbens

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The neural basis of drug craving: an incentive-sensitization theory of addiction

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Persistent Structural Modifications in Nucleus Accumbens and Prefrontal Cortex Neurons Produced by Previous Experience with Amphetamine

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Dissociable deficits in the decision-making cognition of chronic amphetamine abusers, opiate abusers, patients with focal damage to prefrontal cortex, and tryptophan-depleted normal volunteers: evidence for monoaminergic mechanisms

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Enhanced behavioural control by conditioned reinforcers following microinjections of d-amphetamine into the nucleus accumbens.

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6-Hydroxydopamine lesions of the nucleus accumbens, but not of the caudate nucleus, attenuate enhanced responding with reward-related stimuli produced by intra-accumbens d-amphetamine

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Enhanced responding for conditioned reward produced by intra-accumbens amphetamine is potentiated after cocaine sensitization

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A single exposure to amphetamine is sufficient to induce long-term behavioral, neuroendocrine, and neurochemical sensitization in rats.

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Effects of medial prefrontal or anterior cingulate cortex lesions on responding for cocaine under fixed-ratio and second-order schedules of reinforcement in rats.

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The role of excitatory amino acids in behavioral sensitization to psychomotor stimulants

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Role of reward and enhancement of conditioned reward in persistence of responding for cocaine

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7 January 2021

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10.1016/S0006-3223(01)01106-4

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

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