(Q42502127)

1 November 2017

title

Increased mitochondrial superoxide generation in neurons from trisomy 16 mice: a model of Down's syndrome (English)

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1 November 2017

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Schuchmann S

1

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1 November 2017

Heinemann U

series ordinal

2

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1 November 2017

language of work or name

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1 January 2000

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Free Radical Biology and Medicine

1 November 2017

published in

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1 November 2017

volume

28

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1 November 2017

issue

2

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1 November 2017

page(s)

235-250

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Hydroperoxide metabolism in mammalian organs

1 November 2017

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Apoptosis and increased generation of reactive oxygen species in Down's syndrome neurons in vitro

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Free radicals and disruption of neuronal ion homeostasis in AD: a role for amyloid beta-peptide?

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Altered Ca2+ signaling and mitochondrial deficiencies in hippocampal neurons of trisomy 16 mice: a model of Down's syndrome.

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The neuropathology of Alzheimer's disease investigated by transplantation of mouse Trisomy 16 hippocampal tissues

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Thymic abnormalities and enhanced apoptosis of thymocytes and bone marrow cells in transgenic mice overexpressing Cu/Zn-superoxide dismutase: implications for Down syndrome

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Glutamate as a hippocampal neuron survival factor: an inherited defect in the trisomy 16 mouse

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A fine structural study of the hippocampus and dorsal root ganglion in mouse trisomy 16, a model of Down's syndrome

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7 January 2021

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7 January 2021

Metabolism of Oxygen Derivatives in Down's Syndrome

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7 January 2021

Neuronal-specific expression of human copper-zinc superoxide dismutase gene in transgenic mice: animal model of gene dosage effects in Down's syndrome

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Substrate-dependent changes in mitochondrial function, intracellular free calcium concentration and membrane channels in pancreatic beta-cells

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7 January 2021

Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure

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7 January 2021

Normal oxidative damage to mitochondrial and nuclear DNA is extensive

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7 January 2021

10.1016/S0891-5849(99)00226-9

Identifiers

DOI

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1 November 2017

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