(Q38304283)

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Identification of novel hypoxia dependent and independent target genes of the von Hippel-Lindau (VHL) tumour suppressor by mRNA differential expression profiling

scientific article published on December 2000

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Europe PubMed Central

PubMed publication ID

1 September 2017

Identification of novel hypoxia dependent and independent target genes of the von Hippel-Lindau (VHL) tumour suppressor by mRNA differential expression profiling (English)

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

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4

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

Peter J. Ratcliffe

5

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P J Ratcliffe

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

author name string

C C Wykoff

1

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Europe PubMed Central

PubMed publication ID

1 September 2017

C W Pugh

2

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

P H Maxwell

3

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

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publication date

1 December 2000

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Europe PubMed Central

PubMed publication ID

1 September 2017

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

19

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

54

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

6297-6305

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

Overexpression of Stra13, a novel retinoic acid-inducible gene of the basic helix-loop-helix family, inhibits mesodermal and promotes neuronal differentiation of P19 cells.

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Tissue transglutaminase: an enzyme with a split personality

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Hypoxia inducible factor-alpha binding and ubiquitylation by the von Hippel-Lindau tumor suppressor protein

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Hypoxia and mitochondrial inhibitors regulate expression of glucose transporter-1 via distinct Cis-acting sequences

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Mutations of the VHL tumour suppressor gene in renal carcinoma

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Hypoxia induces accumulation of p53 protein, but activation of a G1-phase checkpoint by low-oxygen conditions is independent of p53 status.

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Hypoxia regulates expression of the endothelin-1 gene through a proximal hypoxia-inducible factor-1 binding site on the antisense strand

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Negative regulation of hypoxia-inducible genes by the von Hippel-Lindau protein

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Down-regulation of transmembrane carbonic anhydrases in renal cell carcinoma cell lines by wild-type von Hippel-Lindau transgenes

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Identification of the von Hippel-lindau tumor-suppressor protein as part of an active E3 ubiquitin ligase complex

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The VHL tumour-suppressor gene paradigm

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Endothelin-1 induces vascular endothelial growth factor synthesis in osteoblasts: involvement of p38 mitogen-activated protein kinase

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The von Hippel-Lindau tumor suppressor protein is a component of an E3 ubiquitin-protein ligase activity

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The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

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Hypoxia-inducible factor-1 modulates gene expression in solid tumors and influences both angiogenesis and tumor growth.

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High-throughput tissue microarray analysis to evaluate genes uncovered by cDNA microarray screening in renal cell carcinoma

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HIF-1 alpha is required for solid tumor formation and embryonic vascularization.

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10.1038/SJ.ONC.1204012

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

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0 references

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

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