(Q34327866)

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Chromatin acetylation, memory, and LTP are impaired in CBP+/- mice: a model for the cognitive deficit in Rubinstein-Taybi syndrome and its amelioration.

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15207239%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

Chromatin acetylation, memory, and LTP are impaired in CBP+/- mice: a model for the cognitive deficit in Rubinstein-Taybi syndrome and its amelioration (English)

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15207239%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

Rubinstein-Taybi syndrome

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based on heuristic

inferred from title

cognitive deficit

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based on heuristic

inferred from title

5

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15207239%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

7

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29 December 2019

6

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Eric R Kandel

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15207239%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

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Juan M Alarcón

1

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29 December 2019

Gaël Malleret

2

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29 December 2019

Khalid Touzani

3

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15207239%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

Svetlana Vronskaya

4

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Europe PubMed Central

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29 December 2019

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1 June 2004

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Europe PubMed Central

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29 December 2019

1 reference

Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15207239%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

42

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29 December 2019

6

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29 December 2019

947-959

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29 December 2019

The phosphorylation status of a cyclic AMP-responsive activator is modulated via a chromatin-dependent mechanism

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Abnormal skeletal patterning in embryos lacking a single Cbp allele: a partial similarity with Rubinstein-Taybi syndrome.

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Molecular analysis of the CBP gene in 60 patients with Rubinstein-Taybi syndrome

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Truncated CBP protein leads to classical Rubinstein-Taybi syndrome phenotypes in mice: implications for a dominant-negative mechanism

1 reference

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Rubinstein-Taybi syndrome caused by mutations in the transcriptional co-activator CBP

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Integration of long-term-memory-related synaptic plasticity involves bidirectional regulation of gene expression and chromatin structure

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Acetylation of histone H2B mirrors that of H4 and H3 at the chicken beta-globin locus but not at housekeeping genes

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Diagnostic analysis of the Rubinstein-Taybi syndrome: five cosmids should be used for microdeletion detection and low number of protein truncating mutations

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Phosphorylated CREB binds specifically to the nuclear protein CBP

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Regulation of NF-kappaB by cyclin-dependent kinases associated with the p300 coactivator

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Mitogen-regulated RSK2-CBP interaction controls their kinase and acetylase activities

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Differential activity of progesterone and glucocorticoid receptors on mouse mammary tumor virus templates differing in chromatin structure

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Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins

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Quantitative analysis of CBP- and P300-induced histone acetylations in vivo using native chromatin.

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Histone deacetylase inhibitors and anticancer therapy

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A CBP binding transcriptional repressor produced by the PS1/epsilon-cleavage of N-cadherin is inhibited by PS1 FAD mutations

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Mutations in the kinase Rsk-2 associated with Coffin-Lowry syndrome

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Rolipram, a type IV-specific phosphodiesterase inhibitor, facilitates the establishment of long-lasting long-term potentiation and improves memory

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Histone Deacetylase Inhibition by Sodium Butyrate Chemotherapy Ameliorates the Neurodegenerative Phenotype in Huntington's Disease Mice

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MeCP2 is a transcriptional repressor with abundant binding sites in genomic chromatin

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Control of memory formation through regulated expression of a CaMKII transgene

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Histone acetyltransferase complexes stabilize swi/snf binding to promoter nucleosomes.

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