(Q34204770)

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How calcium influx through calcium leak channels is responsible for the elevated levels of calcium-dependent proteolysis in dystrophic myotubes

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Europe PubMed Central

PubMed publication ID

31 July 2017

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Europe PubMed Central

How calcium influx through calcium leak channels is responsible for the elevated levels of calcium-dependent proteolysis in dystrophic myotubes (English)

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Europe PubMed Central

PubMed publication ID

31 July 2017

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Alderton JM

1

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

Steinhardt RA

2

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Europe PubMed Central

PubMed publication ID

31 July 2017

publication date

1 August 2000

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Europe PubMed Central

PubMed publication ID

31 July 2017

Trends in Cardiovascular Medicine

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Europe PubMed Central

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31 July 2017

10

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Europe PubMed Central

PubMed publication ID

31 July 2017

6

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

268-272

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Europe PubMed Central

PubMed publication ID

31 July 2017

Evidence for a vesicle-mediated maintenance of store-operated calcium channels in a human embryonic kidney cell line

1 reference

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Calcium influx through calcium leak channels is responsible for the elevated levels of calcium-dependent proteolysis in dystrophic myotubes

1 reference

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Deflazacort in Duchenne dystrophy: study of long-term effect

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Delay of muscle degeneration and necrosis in mdx mice by calpain inhibition.

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Photobleaching of fura-2 and its effect on determination of calcium concentrations.

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Calcium-regulated exocytosis is required for cell membrane resealing

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Kinesin- and myosin-driven steps of vesicle recruitment for Ca2+-regulated exocytosis.

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Three muscular dystrophies: loss of cytoskeleton-extracellular matrix linkage.

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Contraction-induced cell wounding and release of fibroblast growth factor in heart

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No alteration in gene expression of components of the ubiquitin-proteasome proteolytic pathway in dystrophin-deficient muscles

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Endogenous inhibitor for calcium-dependent cysteine protease contains four internal repeats that could be responsible for its multiple reactive sites

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Increased activity of calcium leak channels in myotubes of Duchenne human and mdx mouse origin

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Sarcoplasmic reticulum interacts with the Ca(2+) indicator precursor fura-2-am

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Dystrophin abnormalities in Duchenne/Becker muscular dystrophy

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Signs and symptoms of Duchenne muscular dystrophy and Becker muscular dystrophy among carriers in The Netherlands: a cohort study

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A capacitative calcium current in cultured skeletal muscle cells is mediated by the calcium-specific leak channel and inhibited by dihydropyridine compounds

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A critical evaluation of resting intracellular free calcium regulation in dystrophic mdx muscle

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Abnormal calcium homeostasis in Duchenne muscular dystrophy myotubes contracting in vitro

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On a historical note: Duchenne of Boulogne

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Role of transiently altered sarcolemmal membrane permeability and basic fibroblast growth factor release in the hypertrophic response of adult rat ventricular myocytes to increased mechanical activity in vitro

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Elevated subsarcolemmal Ca2+ in mdx mouse skeletal muscle fibers detected with Ca2+-activated K+ channels.

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Loss, restoration, and maintenance of plasma membrane integrity

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Vesicle accumulation and exocytosis at sites of plasma membrane disruption

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Noise-induced transient microlesions in the cell membranes of auditory hair cells

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Aging, cardiac hypertrophy and ischemic cardiomyopathy do not affect the proportion of mononucleated and multinucleated myocytes in the human heart

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Dystrophin protects the sarcolemma from stresses developed during muscle contraction

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Molecular and cellular physiology of intracellular calcium stores

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Calcium-activated neutral protease from muscle and platelets of Duchenne muscular dystrophy cases

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Plasma membrane disruption underlies injury of the corneal endothelium by ultrasound.

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Cell membrane resealing by a vesicular mechanism similar to neurotransmitter release

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Increased protein degradation results from elevated free calcium levels found in muscle from mdx mice

1 reference

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based on heuristic

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Increased calcium influx in dystrophic muscle

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Proteolysis results in altered leak channel kinetics and elevated free calcium in mdx muscle.

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Expression of three calpain isoform genes in human skeletal muscles

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Depression of posttetanic twitch potentiation by low calcium and calcium channel antagonists

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Activation of store-operated Ca2+ current in Xenopus oocytes requires SNAP-25 but not a diffusible messenger

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Identifiers

10.1016/S1050-1738(00)00075-X

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

31 July 2017

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