(Q33994978)

30 July 2017

1 reference

title

Mitochondria as the central control point of apoptosis (English)

1 reference

30 July 2017

1 reference

1 reference

Desagher S

1

1 reference

30 July 2017

Martinou JC

series ordinal

2

1 reference

30 July 2017

publication date

1 September 2000

1 reference

30 July 2017

published in

Trends in Cell Biology

1 reference

30 July 2017

volume

10

1 reference

30 July 2017

issue

9

1 reference

30 July 2017

page(s)

369-377

1 reference

Cell Death in Development

30 July 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Biochemical pathways of caspase activation during apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Cell-free apoptosis in Xenopus egg extracts: inhibition by Bcl-2 and requirement for an organelle fraction enriched in mitochondria.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

An emerging blueprint for apoptosis in Drosophila

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Molecular characterization of mitochondrial apoptosis-inducing factor

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Cytochrome c deficiency causes embryonic lethality and attenuates stress-induced apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Blocking cytochrome c activity within intact neurons inhibits apoptosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

c-Myc-induced sensitization to apoptosis is mediated through cytochrome c release

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

[Research on ultrastructures in an experimental renal tumor in the hamster]

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Chemical and physical fixation of isolated mitochondria in low-energy and high-energy states

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Mitochondrial Proliferation and Paradoxical Membrane Depolarization during Terminal Differentiation and Apoptosis in a Human Colon Carcinoma Cell Line

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Apoptosis, in human monocytic THP.1 cells, results in the release of cytochrome c from mitochondria prior to their ultracondensation, formation of outer membrane discontinuities and reduction in inner membrane potential

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The release of cytochrome c from mitochondria during apoptosis of NGF-deprived sympathetic neurons is a reversible event

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The 55-kDa tumor necrosis factor receptor induces clustering of mitochondria through its membrane-proximal region

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

BCL-2 family members and the mitochondria in apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-2-family proteins: the role of the BH3 domain in apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Regulated targeting of BAX to mitochondria.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-xL Regulates the Membrane Potential and Volume Homeostasis of Mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bax directly induces release of cytochrome c from isolated mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The overexpression of Bax produces cell death upon induction of the mitochondrial permeability transition

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

1 reference

7 January 2021

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Injected cytochrome c induces apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Presence of a pre-apoptotic complex of pro-caspase-3, Hsp60 and Hsp10 in the mitochondrial fraction of jurkat cells

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-xL prevents cell death following growth factor withdrawal by facilitating mitochondrial ATP/ADP exchange

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-2 proteins: regulators of apoptosis or of mitochondrial homeostasis?

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The mitochondrial permeability transition pore and its role in cell death

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Mitochondrial control of apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Fluctuations in mitochondrial membrane potential caused by repetitive gating of the permeability transition pore

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The central executioner of apoptosis: multiple connections between protease activation and mitochondria in Fas/APO-1/CD95- and ceramide-induced apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Apaf1 is required for mitochondrial pathways of apoptosis and brain development

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Distinct stages of cytochrome c release from mitochondria: evidence for a feedback amplification loop linking caspase activation to mitochondrial dysfunction in genotoxic stress induced apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Conversion of Bcl-2 to a Bax-like Death Effector by Caspases

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bcl-2 family proteins as ion-channels

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Solution structure of BID, an intracellular amplifier of apoptotic signaling

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Solution structure of the proapoptotic molecule BID: a structural basis for apoptotic agonists and antagonists

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Ion channel activity of the BH3 only Bcl-2 family member, BID.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Inhibition of Bax channel-forming activity by Bcl-2

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

The Destabilization of Lipid Membranes Induced by the C-terminal Fragment of Caspase 8-cleaved Bid Is Inhibited by the N-terminal Fragment

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Modulation of mitochondrial Ca(2+) homeostasis by Bcl-2.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

Cell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2800%2901803-1

7 January 2021

10.1016/S0962-8924(00)01803-1

Identifiers

DOI

1 reference

30 July 2017

1 reference