(Q33778899)

29 July 2017

title

Truncated beta-amyloid peptide channels provide an alternative mechanism for Alzheimer's Disease and Down syndrome (English)

1 reference

29 July 2017

0 references

1 reference

Ricardo Capone

4

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Srinivasan Ramachandran

object named as

Srinivasan Ramachandran

series ordinal

3

0 references

Fernando Teran Arce

object named as

Fernando Teran Arce

series ordinal

2

0 references

Ruth Nussinov

series ordinal

7

object named as

Ruth Nussinov

1 reference

29 July 2017

1

Hyunbum Jang

1 reference

29 July 2017

6

Bruce L Kagan

1 reference

29 July 2017

8

Ratnesh Lal

1 reference

29 July 2017

Rushana Azimova

5

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29 July 2017

language of work or name

English

0 references

publication date

22 March 2010

1 reference

Proceedings of the National Academy of Sciences of the United States of America

29 July 2017

published in

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29 July 2017

volume

107

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29 July 2017

issue

14

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29 July 2017

page(s)

6538-6543

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Misfolded amyloid ion channels present mobile beta-sheet subunits in contrast to conventional ion channels

29 July 2017

cites work

1 reference

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Models of toxic beta-sheet channels of protegrin-1 suggest a common subunit organization motif shared with toxic alzheimer beta-amyloid ion channels

4 July 2018

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4 July 2018

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P3 peptide, a truncated form of A beta devoid of synaptotoxic effect, does not assemble into soluble oligomers

4 July 2018

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4 July 2018

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4 July 2018

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Models of beta-amyloid ion channels in the membrane suggest that channel formation in the bilayer is a dynamic process

4 July 2018

1 reference

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4 July 2018

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4 July 2018

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4 July 2018

1 reference

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3D structure of Alzheimer's amyloid-beta(1-42) fibrils

4 July 2018

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4 July 2018

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4 July 2018

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Cell biology of protein misfolding: the examples of Alzheimer's and Parkinson's diseases

4 July 2018

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A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse model

4 July 2018

1 reference

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Reconstitution of Cell Membrane Structure in vitro and its Transformation into an Excitable System

4 July 2018

1 reference

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Stabilities and conformations of Alzheimer's beta -amyloid peptide oligomers (Abeta 16-22, Abeta 16-35, and Abeta 10-35): Sequence effects

4 July 2018

1 reference

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Abeta 17-42 in Alzheimer's disease activates JNK and caspase-8 leading to neuronal apoptosis

4 July 2018

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Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

4 July 2018

1 reference

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Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases

4 July 2018

1 reference

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Amyloid beta protein forms ion channels: implications for Alzheimer's disease pathophysiology

4 July 2018

1 reference

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Amyloid-beta peptide fragments p3 and p4 induce pro-inflammatory cytokine and chemokine production in vitro and in vivo.

4 July 2018

1 reference

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Alzheimer's beta-amyloid, human islet amylin, and prion protein fragment evoke intracellular free calcium elevations by a common mechanism in a hypothalamic GnRH neuronal cell line

4 July 2018

1 reference

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Amyloid beta protein (1-40) forms calcium-permeable, Zn2+-sensitive channel in reconstituted lipid vesicles

4 July 2018

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4 July 2018

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4 July 2018

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4 July 2018

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The "nonamyloidogenic" p3 fragment (amyloid beta17-42) is a major constituent of Down's syndrome cerebellar preamyloid

4 July 2018

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4 July 2018

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P3 beta-amyloid peptide has a unique and potentially pathogenic immunohistochemical profile in Alzheimer's disease brain

4 July 2018

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4 July 2018

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A heparin-binding domain in the amyloid protein precursor of Alzheimer's disease is involved in the regulation of neurite outgrowth

4 July 2018

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Chemical characterization of A beta 17-42 peptide, a component of diffuse amyloid deposits of Alzheimer disease.

4 July 2018

1 reference

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The pore dimensions of gramicidin A.

4 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2851998

Alzheimer's disease: the amyloid cascade hypothesis

4 July 2018

1 reference

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Accelerating amyloid-beta fibrillization reduces oligomer levels and functional deficits in Alzheimer disease mouse models.

4 July 2018

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Neurodegenerative disease: amyloid pores from pathogenic mutations

21 January 2018

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Amyloid peptide channels: blockade by zinc and inhibition by Congo red (amyloid channel block).

21 January 2018

1 reference

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Amyloid beta 1-42 deposits do not lead to Alzheimer's neuritic plaques in aged dogs.

21 January 2018

1 reference

https://api.crossref.org/works/10.1073%2FPNAS.0914251107

Amino-terminal Deletions Enhance Aggregation of β-Amyloid Peptides in Vitro

21 January 2018

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https://api.crossref.org/works/10.1073%2FPNAS.0914251107

Pore formation by beta-2-microglobulin: a mechanism for the pathogenesis of dialysis associated amyloidosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1073%2FPNAS.0914251107

Alzheimer's disease. Fresh evidence points to an old suspect: calcium.

21 January 2018

1 reference

https://api.crossref.org/works/10.1073%2FPNAS.0914251107

Designing nanosensors based on charged derivatives of gramicidin A.

21 January 2018

1 reference

https://api.crossref.org/works/10.1073%2FPNAS.0914251107

21 January 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/20308552

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Use of planar lipid bilayer membranes for rapid screening of membrane active compounds

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/20308552

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Amyloidosis and protein folding

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/20308552

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1073/PNAS.0914251107

1 reference

29 July 2017

0 references

1 reference

29 July 2017

1 reference