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The voltage-dependent anion channel: an essential player in apoptosis
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title
The voltage-dependent anion channel: an essential player in apoptosis
(English)
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main subject
voltage
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apoptotic process
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author name string
Yoshihide Tsujimoto
series ordinal
1
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Shigeomi Shimizu
series ordinal
2
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language of work or name
English
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publication date
2002
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stated in
PubMed
published in
Biochimie
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volume
84
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issue
2-3
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page(s)
187-93
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cites work
Breaking the mitochondrial barrier
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
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The mitochondrion in apoptosis: how Pandora's box opens
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reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
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Caspases: enemies within
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reference URL
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7 January 2021
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The Bcl-2 protein family: arbiters of cell survival
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Crossref
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7 January 2021
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Bcl-2 family: life-or-death switch
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7 January 2021
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Translocation of C. elegans CED-4 to nuclear membranes during programmed cell death
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7 January 2021
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Mitochondrial endonuclease G is important for apoptosis in C. elegans.
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
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Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells
1 reference
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Crossref
reference URL
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7 January 2021
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Apoptotic pathways: paper wraps stone blunts scissors
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Crossref
reference URL
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7 January 2021
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Molecular characterization of mitochondrial apoptosis-inducing factor
1 reference
stated in
Crossref
reference URL
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Hsp60 accelerates the maturation of pro-caspase-3 by upstream activator proteases during apoptosis
1 reference
stated in
Crossref
reference URL
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7 January 2021
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Presence of a pre-apoptotic complex of pro-caspase-3, Hsp60 and Hsp10 in the mitochondrial fraction of jurkat cells
1 reference
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Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
Endonuclease G is an apoptotic DNase when released from mitochondria
1 reference
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reference URL
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7 January 2021
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The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4
1 reference
stated in
Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
based on heuristic
inferred from DOI database lookup
Bcl-2 family proteins as ion-channels
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
based on heuristic
inferred from DOI database lookup
Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
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inferred from DOI database lookup
Investigation of bax-induced release of cytochrome c from yeast mitochondria permeability of mitochondrial membranes, role of VDAC and ATP requirement
1 reference
stated in
Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
based on heuristic
inferred from DOI database lookup
A cytochrome c-GFP fusion is not released from mitochondria into the cytoplasm upon expression of Bax in yeast cells
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c.
1 reference
stated in
Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
BAX-dependent transport of cytochrome c reconstituted in pure liposomes
1 reference
stated in
Crossref
reference URL
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7 January 2021
based on heuristic
inferred from DOI database lookup
Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
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inferred from DOI database lookup
Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity
1 reference
stated in
Crossref
reference URL
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7 January 2021
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Colicin Pore-Forming Domains Bind toEscherichia ColiTrimeric Porins†
1 reference
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inferred from DOI database lookup
Biochemical and genetic analysis of the mitochondrial response of yeast to BAX and BCL-X(L)
1 reference
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Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
Role of oxidative phosphorylation in Bax toxicity.
1 reference
stated in
Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells
1 reference
stated in
Crossref
reference URL
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7 January 2021
based on heuristic
inferred from DOI database lookup
Apoptosis control in syncytia induced by the HIV type 1-envelope glycoprotein complex: role of mitochondria and caspases.
1 reference
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Crossref
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Mitochondrial porin required for ischemia-induced mitochondrial dysfunction and neuronal damage
1 reference
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Crossref
reference URL
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inferred from DOI database lookup
Inhibition of Bax channel-forming activity by Bcl-2
1 reference
stated in
Crossref
reference URL
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7 January 2021
based on heuristic
inferred from DOI database lookup
Evidence for secretory pathway localization of a voltage-dependent anion channel isoform
1 reference
stated in
Crossref
reference URL
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7 January 2021
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inferred from DOI database lookup
Studies on human porin XXI: gadolinium opens Up cell membrane standing porin channels making way for the osmolytes chloride or taurine-A putative approach to activate the alternate chloride channel in cystic fibrosis
1 reference
stated in
Crossref
reference URL
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7 January 2021
based on heuristic
inferred from DOI database lookup
Inhibition of apoptosis by the actin-regulatory protein gelsolin
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
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inferred from DOI database lookup
Gelsolin inhibits apoptosis by blocking mitochondrial membrane potential loss and cytochrome c release
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
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inferred from DOI database lookup
Gelsolin in complex with phosphatidylinositol 4,5-bisphosphate inhibits caspase-3 and -9 to retard apoptotic progression
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Human gelsolin prevents apoptosis by inhibiting apoptotic mitochondrial changes via closing VDAC
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
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7 January 2021
based on heuristic
inferred from DOI database lookup
Hepatitis B virus X protein colocalizes to mitochondria with a human voltage-dependent anion channel, HVDAC3, and alters its transmembrane potential
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Neisseria meningitidis porin PorB interacts with mitochondria and protects cells from apoptosis
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Targeting of the pro-apoptotic VDAC-like porin (PorB) of Neisseria gonorrhoeae to mitochondria of infected cells
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
VDAC regulation by the Bcl-2 family of proteins
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The mitochondrial permeability transition.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Mitochondrial control of cell death
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0300-9084%2802%2901370-6
retrieved
7 January 2021
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inferred from DOI database lookup
Identifiers
DOI
10.1016/S0300-9084(02)01370-6
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
417115
OpenCitations bibliographic resource ID
417115
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
417115
PubMed publication ID
12022949
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
417115
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