(Q24297605)

Journal of Molecular Biology

GOA release 2020-03-11

1 reference

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2

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Matthew McKenzie

series ordinal

1

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David Thorburn

object named as

David R Thorburn

series ordinal

3

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Michael T Ryan

series ordinal

4

object named as

Michael T Ryan

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language of work or name

English

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publication date

18 August 2006

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published in

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volume

361

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issue

3

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page(s)

462-9

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cites work

X-linked cardioskeletal myopathy and neutropenia (Barth syndrome): an update

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X-linked cardioskeletal myopathy and neutropenia (Barth syndrome): respiratory-chain abnormalities in cultured fibroblasts

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Tricine-sodium dodecyl sulfate-polyacrylamide gel electrophoresis for the separation of proteins in the range from 1 to 100 kDa

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Blue native electrophoresis for isolation of membrane protein complexes in enzymatically active form

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An X-linked mitochondrial disease affecting cardiac muscle, skeletal muscle and neutrophil leucocytes

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Phospholipid abnormalities in children with Barth syndrome

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The biosynthesis and functional role of cardiolipin

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Barth syndrome may be due to an acyltransferase deficiency

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Respiratory complex III is required to maintain complex I in mammalian mitochondria

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Photolabeling of cardiolipin binding subunits within bovine heart cytochrome c oxidase

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Interactions between phospholipids and NADH:ubiquinone oxidoreductase (complex I) from bovine mitochondria

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Phospholipase digestion of bound cardiolipin reversibly inactivates bovine cytochrome bc1.

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Phospholipase A(2) digestion of cardiolipin bound to bovine cytochrome c oxidase alters both activity and quaternary structure

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Characterization of lymphoblast mitochondria from patients with Barth syndrome

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Cytochrome c acts as a cardiolipin oxygenase required for release of proapoptotic factors

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A novel X-linked gene, G4.5. is responsible for Barth syndrome

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Cardiolipin deficiency in X-linked cardioskeletal myopathy and neutropenia (Barth syndrome, MIM 302060): a study in cultured skin fibroblasts

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Gluing the respiratory chain together. Cardiolipin is required for supercomplex formation in the inner mitochondrial membrane

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Insertion and assembly of human tom7 into the preprotein translocase complex of the outer mitochondrial membrane

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Only one splice variant of the human TAZ gene encodes a functional protein with a role in cardiolipin metabolism

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Cardiolipin stabilizes respiratory chain supercomplexes

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Cardiolipin biosynthesis and mitochondrial respiratory chain function are interdependent

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Significance of respirasomes for the assembly/stability of human respiratory chain complex I.

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The human TAZ gene complements mitochondrial dysfunction in the yeast taz1Delta mutant. Implications for Barth syndrome

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Cardiolipin is essential for organization of complexes III and IV into a supercomplex in intact yeast mitochondria

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Respiratory chain supercomplexes

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The X-linked gene G4.5 is responsible for different infantile dilated cardiomyopathies.

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Taz1, an outer mitochondrial membrane protein, affects stability and assembly of inner membrane protein complexes: implications for Barth Syndrome

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Supercomplexes in the respiratory chains of yeast and mammalian mitochondria

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Specific roles of protein-phospholipid interactions in the yeast cytochrome bc1 complex structure

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Monolysocardiolipins accumulate in Barth syndrome but do not lead to enhanced apoptosis

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